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which is, stimulation of CD74 with exogenous MIF may well bring on an excessive inflammatory condition, as MIF is effective at stimulating CXCR2 and CXCR4 receptors Together with CD74. CXCR2 and CXCR4 receptors when activated encourage influx of neutrophils and lymphocytes, respectively (63, 64).
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A Observe of warning is needed here nevertheless, for the reason that MIF and D-DT are important for progress of many alternative progenitor cells [132, 142, 143]. Regardless that MIF has shown protective consequences on inflammation-induced harm, antagonism of MIF actions may not be ideal when dest